Journal of neuroinflammation

Age exacerbates the CCR2/5-mediated neuroinflammatory response to traumatic brain injury.

Age exacerbates the CCR2/5-mediated neuroinflammatory response to traumatic brain injury.

NF-?B transcriptional activation by TNFa requires phospholipase C, extracellular signal-regulated kinase 2 and poly(ADP-ribose) polymerase-1.

NF-?B transcriptional activation by TNFa requires phospholipase C, extracellular signal-regulated kinase 2 and poly(ADP-ribose) polymerase-1.

Recurrent/moderate hypoglycemia induces hippocampal dendritic injury, microglial activation, and cognitive impairment in diabetic rats.

Recurrent/moderate hypoglycemia induces hippocampal dendritic injury, microglial activation, and cognitive impairment in diabetic rats.

Tumor necrosis factor-a synthesis inhibitor 3,6'-dithiothalidomide attenuates markers of inflammation, Alzheimer pathology and behavioral deficits in animal models of neuroinflammation and Alzheimer's disease.

Tumor necrosis factor-a synthesis inhibitor 3,6'-dithiothalidomide attenuates markers of inflammation, Alzheimer pathology and behavioral deficits in animal models of neuroinflammation and Alzheimer's disease.

Microglial activation induced by brain trauma is suppressed by post-injury treatment with a PARP inhibitor.

Microglial activation induced by brain trauma is suppressed by post-injury treatment with a PARP inhibitor.

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