Neuroscience

Hyperalgesic priming is restricted to isolectin B4-positive nociceptors.

State-dependent plasticity of the corticostriatal pathway.

State-dependent plasticity of the corticostriatal pathway.

Nociceptor subpopulations involved in hyperalgesic priming.

Triggering genetically-expressed transneuronal tracers by peripheral axotomy reveals convergent and segregated sensory neuron-spinal cord connectivity.

Triggering genetically-expressed transneuronal tracers by peripheral axotomy reveals convergent and segregated sensory neuron-spinal cord connectivity.

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

Dependence of monocyte chemoattractant protein 1 induced hyperalgesia on the isolectin B4-binding protein versican.

Contributions of the amygdala and medial prefrontal cortex to incentive cue responding.

Synaptic vesicle protein trafficking at the glutamate synapse.

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